lipoatrophy. typical "bra-wearer" lipoatrophy. this band-like lipoatrophy is caused by tightly fitting bra straps and is to be interpreted as pressure atrophy. the duration of these depressions in the patient's shoulder area is unknown. no complaints. the overlying skin is unaffected.
Acrodermatitis chronica atrophicans: Symptoms existing for 1 year with an acral accentuated, inhomogeneous, blurred, edematous, red, rough swelling on the back of the right foot and extending to the lower leg in a 70 year old female patient.
Acrodermatitis chronica atrophicans. cigarette-paper-like wrinkles on the edematous swollen, flat erythematous right foot. a broad erythematous stripe ran along the tibia. the patient reported pain and instability while walking, but a neuroborelliosis was excluded.
Acrodermatitis chronica atrophicans. Clearly visible, flaccid skin atrophy and edematous redness on the right foot in a serologically proven infection with Borrelia bacteria. The patient spends several months every summer in the Black Forest.
acne conglobata. multiple comedones in the area of nose, cheeks and neck of a 53-year-old patient. irregular skin surface with pronounced scarring, predominantly deeply indented. approx. 2 cm large hyperpigmentation at the root of the nose.
Acne conglobata. multiple comedones in the area of the back of a 53-year-old patient with A. conglobata since the age of 14. no papules or pustules. irregular skin surface with pronounced scarring. coin-sized, deep scar (almost central in the picture) after incision of an abscess.
actinomycosis (abdominal form). progressive fistulizing clinical picture in a 50-year-old patient since several years. the left half of the buttocks was infiltrated in a flat, board-like manner. no significant pain. besides blue-red coarse scarring, granulation tissue and fistulas with exudate (buttock center, Rima ani) are impressive.
anetodermia. disseminated, 0.3-1.0 cm large, whitish, roundish to oval, completely symptomless "scar-like" foci with atrophically curled skin. in places the HVs also bulge over the skin surface. the HVs have existed for several years and were subjectively perceived as disturbing.
Anetodermia. inflammatory anetodermia (type Jadassohn). 0.5 cm to 1.5 cm large, reddish-brownish, centrally atrophic sunken, roundish to oval HV with increased foldable skin in the area of the neck. HV exist for about 3 years. no subjective complaints. clinical picture still progressive!
Angiokeratoma of the glans penis. multiple, chronically stationary, 0.2-0.4 cm large, blue-red to brownish papules with partly smooth, partly scaly surface in the area of the corona glandis. these are congenital, circumscribed vascular ectasias.
Aplasia cutis congenita. a sharply defined, whitish-yellowish, shallowly sunken, hairless area with atrophic, parchment-like skin surface, existing since birth. on incident light microscopy any follicular structures are missing.
Aplasia cutis congenita. detail magnification: Classically specular, slightly sunken, light yellow macula with smooth surface. in the marginal area, in the lower left picture, a smaller area with tufted hairs is visible.
Aplasia cutis congenita. general view: 1.5 x 1.2 cm in size, hairless, slightly sunken, yellowish-whitish area with shiny surface, existing since birth, unchanged for years except for size increase during physical growth.
atrophy. 63-year-old female patient with known porphyria cutanea tarda. evenly tanned facial skin. vertically running, sharp wrinkles of the forehead skin as well as deep, radial wrinkles of the perioral region. involution of the lip red.
Atrophy: Flaccid atrophy (Cutis rhomboidalis nuchae) in an 82-year-old farmer. Solar atrophy of the skin of the neck and the lateral parts of the neck. Paving stone-like surface relief with intersecting longitudinal and oblique furrows (Cutis rhomboidalis nuchae), caused by the severe elastosis actinica (typical is the yellow-white colour of the skin change). The atrophic furrow formation ends abruptly in the area of the hairline (UV-protective effect).
Atrophy.striae cutis distensae.53-year-old patient who has been treated externally and internally with glucocorticoids for one year. striae cutis distensae without subjective symptoms. 2-3 mm wide flat atrophic lesions running in transverse direction to the skin tension with a parchment-like surface. the red tone is caused by the rupture of the connective tissue and atrophy of the surface so that vessels can shine through.
Atrophie blanche. reflected light microscopy (section of a lesion on the lower leg): Atrophic-diluted, transparent epidermis with subepidermal, meander-like tangled vascular convolutes and blood leaks of different ages; whitish opaque, unstructured skin areas (fibrotic connective tissue) are also visible.
Please login to access all articles, images, and functions.
Our content is available exclusively to medical professionals. If you have already registered, please login. If you haven't, you can register for free (medical professionals only).
Please complete your registration to access all articles and images.
To gain access, you must complete your registration. You either haven't confirmed your e-mail address or we still need proof that you are a member of the medical profession.