Chronic light damage: poikiloderma after years of excessive UV exposure, including hyperpigmentation, depigmentation and numerous precanceroses of the actinic keratosis type.
Type I Neurofibromatosis, peripheral type or classic cutaneous form Peripheral neurofibromatosis with multiple skin-coloured to light brown, soft nodes and nodules, sometimes also stalked, bulging soft, skin-coloured dewlap on the left hip.
Usual melanocytic nevus. Chronic stationary, 0.2-2.0 cm in size, sharply defined, soft, light to dark brown, indolent, smooth papules and plaques in the breast area of a 22-year-old woman. No increase in pigmentation or size is noticeable.
Dyskeratosis follicularis. partly loosely disseminated, partly grouped standing, partly aggregated (sternal region), brown-red papules and plaques in a 52-year-old female patient who suffered from "eczematous" changes in the seborrhoeic zones since early childhood. distinct itching, increased body odour. a worsening of the disease over several weeks led to this skin condition, with evidence of Candida spp. and Staph. aureus in smears.
type I neurofibromatosis, peripheral type or classic cutaneous form. numerous smaller and larger soft papules and nodules. several so-called café-au-lait spots.
type i neurofibromatosis, peripheral type or classic cutaneous form. numerous smaller and larger soft, predominantly pigmented, practical nodules and nodules. in the larger nodules the so-called "bell-button phenomenon" can be detected. the palpating finger penetrates the deep dermis as if through a fascial gap. few café-au-lait spots. papules and nodules. only isolated rather discreet café-au-lait spots.
Type I neurofibromatosis, peripheral type or classic cutaneous form. massive tumorous transformation of the skin with numerous generalized distributed, soft, skin-colored, partly pointed conical shaped neurofibromas on the left mamma. the CT examination (skull) did not reveal any pathological findings. no neurofibromas known in the family.
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