Immunodeficiency 38 D84.8

Last updated on: 12.04.2022

Dieser Artikel auf Deutsch

Requires free registration (medical professionals only)

Please login to access all articles, images, and functions.

Our content is available exclusively to medical professionals. If you have already registered, please login. If you haven't, you can register for free (medical professionals only).


Requires free registration (medical professionals only)

Please complete your registration to access all articles and images.

To gain access, you must complete your registration. You either haven't confirmed your e-mail address or we still need proof that you are a member of the medical profession.

Finish your registration now

DefinitionThis section has been translated automatically.

Autosomal recessive immunodeficiency 38 (IMD38; see also Mendelian susceptibility to mycobacterial diseases) is a rare immunodeficiency syndrome caused by a mutation in the ISG15 gene (ISG15 stands for "ubiquitin like modifier"). ISG15 is a protein-coding gene located at chromosome 1p36.33. The protein encoded by this gene is a ubiquitin-like protein that attaches to intracellular target proteins upon activation by interferon-alpha and interferon-beta(ubiquitination).

The autosomal recessive ISG15 mutation causes a specific immune deficiency that results in severe clinical disease in affected individuals when infected with low virulence mycobacteria including Mycobacterium bovis Bacille Calmette-Guerin (BCG) vaccines (Bogunovic et al. 2012). In contrast, there is no immune deficiency to viral infections. Affected individuals exhibit intracranial calcification (Zhang et al. 2015).

LiteratureThis section has been translated automatically.

  1. Bogunovic D et al. (2012) Mycobacterial disease and impaired IFN-gamma immunity in humans with inherited ISG15 deficiency. Science 337: 1684-1688.
  2. Zhang X et all. (2015) Human intracellular ISG15 prevents interferon-alpha/beta over-amplification and auto-inflammation. Nature 517: 89-93.

Last updated on: 12.04.2022